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The putrefactive process as the cause of disease and death and microbe-like
formations in the blood of chronically ill people*
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By
Dr Erik O. H. Enby, MD, Göteborg 1994.
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Abstract
In the lecture delivered at the International Cancer Congress on
16-18 April 1994 in Darling Harbour, Sydney, Australia, the states
of diseases in general are explained as a consequence of different
forms of growing processes in blood and solid tissues and how these
processes continue in the soma after the moment of death. They are
supposed to cause both disease and putrefaction. A number of growing
principles is demonstrated.
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Introduction
We are in my office in Gothenburg with my patient Beverly, who has
breast cancer. Tumours have been found in her right breast. She refused
any orthodox treatment except surgery, which was performed about a
year ago. Now she has new tumours in the remaining parts of her breast
and in the scar. I gave her various alternative treatments. The tumour
changed and became weak in its consistency. I was able to press parts
of the tumour out of her breast manually.
At once I took a little piece of this cancer substance, put it on
a slide, placed another slide over it, and pressed the two slides
very hard between my fingers. Thus I could press out the cancer substance,
getting a very thin layer between the two glasses, and examine the
histopathology of the sample microscopically.
The cancer substance seemed to consist of two parts. Lots of moving
granules of different sizes, and a kind of cheese-like substance
in which the granules were also seen. I was really most astonished
when I saw the moving granules, because looking at them did not
make me think of cancer cells. They represented something else.
However, when I looked very carefully at the cheese-like substance,
I could see that it consisted of rather large cells about 15-25
µm in diameter, the contours of which were almost totally
rubbed out. Among these cells there were also lots of very thin
filament-like structures going through the specimen in all directions.
It was possible to see these filaments better if the preparation
was influenced by different chemical substances.
The granules in the cheese-like substance did not move, probably
because they were mixed up with the substance and pressed so hard
between the glasses that they couldn’t move. The tumour substance
was sent to the pathology lab at the University of Gothenburg, and
the examination showed the histological picture of a highly malignant
tumour.
To find an effective treatment for Beverly was until now not possible.
In the following statement I will try to remind the audience how
important it is to make a correct interpretation of any histopathological
finding, in order to understand its deepest nature, so that an adequate
therapy can be chosen and random treatments avoided. That is, to
understand from the histopathological picture, the very abstract
processes in a tissue that cause normal histology to change into
pathohistology, resulting in a somatic disturbance with a secondary
disease picture, conditio sine qua non
to be able to find a cause-blocking remedy to hamper the propagation
of the somatic disturbance, in order to stop the development of
or to eradicate the disease picture. In the following I am going
to speak about growth as a plausible cause.
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Growth
Growth implies a relationship between a growth product and a soil
and the growth process in the product. Growth can only continue
if there is a feeding soil in close relation to the growing product.
Consequently, we can also maintain that nothing can grow in itself
because a growth product cannot function as a feeding soil to itself
for its own growth. That could be an axiom. Now, soil is not only
feeding the growing growth product, but it is also impoverished
by it. From this point of view, the growth process and the growth
product are always dominating the soil, and the growth will not
stop as long as the soil is delivering nourishment. From this philosophic
discussion and the fact that tumours are actually growing in the
body, we can extrapolate that a growing tumour also needs its soil
(the tissues) to be able to grow, and that a cancer substance is
something quite different from the tissues within which it is growing.
It is not possible to postulate that it is emanating from normal
tissue cells that changed their behaviour.
In Beverly’s case, as you will understand later from this
lecture, there are strong reasons to suspect that the granules and
the filaments in her cancer substance may show a pathogenic fungus
infection. My hypothesis is that the granules represent the yeast-phase
of this fungus. Budding and multiplying locally, they create the
growing ”cancer substance” in the breast tissue. This
kind of postulated parasitic growth will eventually change the tissue
in its microstructure and metabolism as well as physically. Many
of these granules will also spread out into the body fluids. Budding
and multiplying at the expense of the body, they will eventually
change and impoverish the tissues, as growth throughout nature reduces
the nourishment contents of the soil towards poverty.
The more intensive a growth is, the more inconvenience the body
will experience. This suffering most often ends with death. The
inner functions of the individual cannot be fettered to the soma
if this kind of growth process damages it too much, but leaves it.
From the linguistic aspect, we could note that ”to die is
to leave the life behind oneself”.
The destruction of the soma by a growth process will therefore continue,
after this moment - according to the dominance over the soil - and
eventually the growth process will transform all tissues to something
quite different. The end result of this transformation and growth
process can be studied in old - normally putrefied corpses.
Consequently, I’m trying to say that growth processes can
start in the soma and are responsible for both the disease and putrefactive
processes, which basically represent the same thing - the somatic
destruction. Understanding these growth processes in order to learn
to hamper them (or better still stop them) must be conditio
sine qua non - to define a treatment that would make a diseased
individual experience a feeling of coming over from the state of
disease to the state of health.
Professor Enderlein of Germany gave me the impulse to think about
disease in this way, and he maintained that the whole life process
(any tissue) contained special living potentials (vegetations) that
suddenly could become able to grow within and destroy our bodies.
Because I had never before heard about these vegetations during
my whole medical career, I decided to check what Professor Enderlein
had seen in the microscope, and to try to find these vegetations
in the body fluids and tissues of chronically ill individuals and
in corpses. This study made me understand that Enderlein stated
something very interesting when he declared that different kinds
of growing vegetations in the soma can cause both the disease and
putrefactive processes. He also stated that these vegetations seem
to be more aggressive after death, when the situation in the corpse
is totally calm, which means that the circulation will not supply
them with nourishment any more. According to the dominance of a
growth product over its soil, they will now, more and more, increase
their growth in all the different tissues of the soma, at much greater
speed than before death.
I am now going to show you some of these vegetations and also give
you some examples of interpreting them, through comparing them with
already well-known facts in the accepted established microbiology.
To begin looking at blood microscopically means that the researcher
will become aware of more or less a jungle of different structures
and particles, spread out in the plasma among the red and white
blood cells. Blood from individuals with different chronic diseases
does not show the same microbe-like formations and growing structures,
and this will eventually make an observer believe that they in some
way must be interlaced with the disease process. To see all these
things for the first time also means that it is difficult for an
observer to relate them to already well-known conditions, which
of course is necessary in order to be able to define their relative
role and importance in the body. I myself could, to begin with,
only with certainty maintain that what I saw in the microscope until
now had not been described in the ordinary medical books.
To make a very first interpretation of these structures, I decided
to find out if something similar to these life forms was described
in the already established microbiological literature. This seemed
to give a very first relative understanding of some probably unknown
structures, that already might have been described in the excretions,
and infected solid tissues of individuals attacked by different
pathogenic fungi and actinomycetes found in human beings. First,
however, I will show the cheese-like substance from Beverly’s
cancer growth. It exposed thread-like structures going through the
specimen in all directions (Figure
1). Similar filament-like structures were also found in the
living blood, visible through the configuration of the erythrocytes
(Figure 2). The flame-like pattern
shows that there is something mixed up with the blood - probably
growing.
Why do I think so? First of all, not only because a structure is
not there a priori, it must have been formed or developed, but also
because these filaments can show a budding growth of small particles.
This might show the transition of a mycelial growth to the yeast-like
form of the same fungus (Figure 3).
Similar filaments can also be fragmented into small particles, and
it is common that this happens among the species in the order of actinomycetales
in the plasma (Figure 4). If only
a few particles can be seen in the plasma, it is impossible to get
any understanding of them. But as soon as you get an idea how they
are created - here from the filament - you will at once think that
these structures might be associated with a kind of actinomycotic
infection (Figure 5).
This very strange thing was impossible for me to connect to anything
in microbiology that I knew. I found round white cavities in the
blood-smear. Large numbers of moving, microbe-like formations of
different shapes and sizes always appear in the cavities. The cavities
may be bubble-like formations. They occur singly, but also in large
numbers, and are often held together with band-like threads that
run between the blood-corpuscles in the smear.
In one patient - a new-born child with Down’s syndrome - I
found very, very big bubbles with large numbers of moving particles
(Figure 6). The plasma around
the bubbles was almost clean. So, these bubbles seem to be filled
with microbe-like particles. It is remarkable that children with
Down’s syndrome eventually get rid of these contaminations
in their blood. The blood clears up and later in life the blood
looks normal. Perhaps this kind of infection damages the soma very
easily, leading to a diffuse somatic impairment. Later development
and growth of the body towards these definite impairments of the
fetal soma might result in the characteristic look of these children.
In Figure 7 you can see a strange
structure, revealed by the configuration of the red blood cells.
It can be described as a disc consisting of an opaque substance,
surrounded by a light corona zone in which lots of moving, microbe-like
formations are found. In the blood, this structure must be floating
around freely as a ball-like structure, with an infectious potential
at its periphery. If observed in the same patient during a long
period, it is possible to find that they can increase in size. It
is easy to understand what this might mean to the body on different
levels. Of course, it is possible that these ball-like formations
can grow also in a solid tissue, and this would then eventually
create a resistance.
In the literature, it is possible to find similar structures, described
as the so-called ”sulphur granules”. They can be found
in solid tissues infected by the pathogenic actinomycetes and contain
an opaque mass in the centre.
In Figure 8 you can see strange
bright areas without erythrocytes on this slide. These have no clear
border on the surrounding blood cells.
Likewise, even if an area looks empty, it almost always shows something
similar to an infection, with many different types of microbe-like
formations. If there are some red blood cells in these areas, they
are partly destroyed and look moth-eaten (Figure
9). You can also see filament-like structures that have been
fragmented. When you look at this, and if you read about the pathogenic
actinomycetes, you will probably suspect that this infection might
belong to this family. It is easy to realize what will happen if
different solid tissues are attacked by these suspected infections
in the same way.
In 1984, I found something that I named a ”flake”. At
first I believed it was of no interest on the glass, but then I
saw that such a flake suddenly was able to produce lots of small
granules, and that these granules were budding and growing, and
changing their form (Figure 10).
All this was possible to observe on the slide in the microscope
during a week. The flakes were similar to the so-called ”grains”,
which are common in fluids coming from tissues infected with pathogenic
fungi and the pathogenic actinomycetes. These grains can be found
in biopsy material, in pus, and on gas bandages. They are up to
0.5 mm in size. In the literature, they are described as a product
of intertwined filaments. With the interference contrast microscope
it was, in fact, sometimes possible to see that the flakes found
in the blood also showed and contained filament-like materials.
It is interesting to speculate on the possibility that the ”filaments”
of these flakes represent the mycelial growth phase of a pathogenic
fungus, or the filamental growth of a pathogenic actinomycete, and
that the sudden production of granules from these flakes represents
the budding yeast-phase of a fungus or a transition of actinomycetic
filaments into these granules.
In Figure 11 you can observe
a little flake in the middle of a heap of granules probably produced
by and from this flake. Once the budding process has started, it
goes on by itself, and in the blood you will then find lots of heaps
of roe-like formations floating around, perhaps creating thrombose-like
states of disease. It is easy to realize that budding of these granules
in solid tissues could create a resistance - a tumour - as in Beverly’s
breast-tissue.
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Conclusion
My information here showed only the tip of an iceberg. As you already
understand, it is not a task for a single person to map out and interpret
the role of these different vegetations in a body. To implement this,
there must be an institutionalization of this field of research within
the science of pathology.
To understand how these vegetations are growing and destroying the
body, will eventually help us to develop a biological concept of
disease according to the biological phenomenon that it is.
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© 1994-2004. Dr Erik Enby. All rights reserved. This article
may only be reproduced in its entirety.
*This paper was first published in 1994
in "Proceedings
of the
1st World Congress on Cancer" under the title The
Decay Process As the Cause of Disease and Death
and Microbial Formations in the Blood of
Chronically Diseased. Independent Medical Research,
Sydney,
Australia.
ISBN 0 646 25093 0.
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